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EXAMPLES FROM CORPUS
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Abnormalities it, urinary but not intestinal excretion of uric acid may produce clinically recognizable disorders of urate metabolism.
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After several months of therapy with a uricosuric agent, the tissue stores are dePleted and urinary uric acid returns to normal.
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Birds excrete uric acid rather than urea because it is an insoluble solid.
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Dietary purine intake may provide a significant source of uric acid.
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Essential hypertension is often associated with an augmented proximal reabsorption of sodium and uric acid.
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Thus, alterations of either uric acid filtration, reabsorption or secretion may result in clinical disorders.
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Uricosuric agents generally do not lower uric acid levels below the normal range.
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Xanthinuria is a rare hereditary deficiency of xanthine oxidase, which results in hypouricemia and diminished urinary uric acid excretion.